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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enargement without pills vimax penis enlargement information penis enargement without pills vimax penis enlargement exercise magna rx testimonials penis enlagement pump penis elargement surgery cost penis enlagement tool

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Almost all human beings are conscious about their beauty and they adopt all means to improve or maintain it. The quality of skin is having priority in the field of beauty care. Skin is considered as the mirror of the human body and it reflects the physical, mental and emotional status of every person. The number of people attending beauty clinics and dermatologists is increasing day by day. Even though skin diseases are having low mortality, people are worried about skin problems because of the social stigma. A person having a disease of some internal organ may not be worried but another person having a skin disease on the face may be depressed because of the simple reason 'others will come to know about my disease'. Human skin has got several functions like protection, heat regulation, water balance, excretion etc. It gives protection and support to the deep body tissues and has got the ability to stretch to some extent due to the presence of protein fibers in the dermis. But too fast enlargement of the body parts cause excessive stretching on the skin resulting in breaking of collagen and elastin fibers of the dermis. This causes slightly depressed discoloration called stria atrophia or stretch marks. Initially these marks are slightly pinkish and later become white or silvery in appearance. Stretch marks are found in conditions like pregnancy, puberty, obesity etc. It is usually seen in places like abdomen, thighs, breasts, buttocks, shoulders etc. Once these marks appear, time is required for them to be less prominent. Both men and women are the victims of stretch marks but it mainly affects women due to their beauty consciousness and causes psychological trauma leading to depression. On the other hand there are women who are least bothered to get rid of stretch marks and consider them as a badge of mother hood. Still the number of people visiting clinics to remove stretch marks is increasing day by day. Because of the increasing need for the stretch mark treatment modalities, there are number of products in the market to deal with stretch marks cure. But most of those products are not proved effective scientifically but few of them give results in many cases. Modern treatment methods like plastic surgery and laser surgery for stretch marks have been proved effective in the treatment. penis enlargment drug blood erection vimax penis pills penis enargement forum penis enhancement product best penis elargement surgery vimax buy penis enlargement pills com enlarement penis penis pump penis enlarement surgery cost penile enlargement surgery cost

Personal relationships can be seriously compromised by a continuing sexual problem. Such sexual dysfunction can cause terrible distress and can disrupt or even spell the end of personal relationship, regardless of which partner has the problem. Sexual dysfunction may be caused through physical problems but anxiety will often aggravate the dilemma. Sexual relationships are never entirely simple but they are very important and a source of much happiness for those in long term relationships. Many things, both physical and psychological, can go wrong and can threaten the fibre of the relationship if not dealt with in a proper manner. It is important for people to have some knowledge of what can impact on failure to achieve satisfactory sexual fulfillment. Such things can be caused by physical problems on the part of either partner or may be psychosomatic. Whatever the case, the problem affects both partners as such a relationship involves intense emotions and other mental factors. Factors such as faulty expectations, poor communication of sexual needs, ignorance, and concern over ability to perform can affect sexual function and satisfaction. Male Sexual Dysfunction Male dysfunction is most commonly in the form of the inability to achieve an erection or the inability to maintain an erection sufficiently to allow normal intercourse. This condition is known as impotence and can cause great distress to the male, not only because it prevents satisfying sexual intercourse but also because many men think it indicates a lack of masculinity. Most men suffer episodes of impotence at some time and these episodes are almost always of a psychological origin. Very few are attributable to disease and those cases that are, are usually among older men. Psychogenic impotence happens quite often because of performance anxiety. However, the majority of women do not place a great deal of importance on the occasional episode of impotence and are usually sympathetic and understanding rather than critical of their partner. They do not normally see it as a deficiency in the man’s masculinity. Sometimes, organic impotence can be helped by drugs like Viagra. In fact, it was only when Viagra was introduced to the market, the true prevalence of erectile dysfunction was revealed. Premature ejaculation, as its name implies, is when the male orgasm happens too early, thus depriving both partners of sexual satisfaction. This can even happen before penetration and is normally due to excessive excitement. This is fairly common in inexperienced men but will settle down as they become more sexually skilled. There is also a condition called Priapism that is potentially dangerous to the man. It is a rare condition in which the erection does not subside after he reaches orgasm. It is important that he seek immediate treatment as the blood in the penis will usually clot after about four hours, forming damaging internal scar tissue. The condition is usually treated by draining the blood under anaesthesia. Priapism has been known to be caused by drug abuse. Another disorder of the penis is Peyronie’s disease of which the cause is unknown. This disorder is characterized by a thickening and rigidity of tissue, resulting in a bend in the penis on erection. This can interfere with normal intercourse by causing discomfort to both partners. It may also prevent sexual intercourse from happening at all. The condition is often helped by steroid injections but surgical removal of the thickened areas is usually needed. Female Sexual Dysfunction Due to unrealistic expectations, many men see women who fail to achieve orgasm as being frigid. However, this often occurs because of a lack of affectionate expression by the partner, or a lack of sexual understanding and skill. Of course, there are other causes such as fear of pregnancy, recent childbirth, dyspareunia (pain during intercourse), and some prescription drugs. Drugs prescribed to treat conditions such as depression, insomnia, or high blood pressure can prevent female orgasm. Approximately ten percent of women will never achieve orgasm and around half never experience orgasm during sexual intercourse due to insufficient foreplay. Men often see the lack of female orgasm as a criticism of their own masculinity. Additional Sexual Problems Dyspareunia is the medical terminology for painful sexual intercourse which may be of physical or psychological origin. For instance, a woman who has recently had an episiotomy repair following childbirth will suffer from dyspareunia if she engages in sexual intercourse too soon. It may also be caused by infections in the uterus or the vagina or from rare congenital defects in the vagina. Pain can also be psychological and can be experienced because of fear or anger. It can also be an instinctive tactic to avoid unwanted sex. There is also an extreme condition called vaginismus which is an involuntary rejection of sexual intercourse and is difficult to treat. Sexual Therapy Those who suffer from any of the conditions mentioned may benefit from a referral to a therapist who will discuss treatment and options. Therapy can help couples overcome their fears of communicating sexual needs and their fear of rejection by their partner by using behavior therapy such as sensate focusing. This is generally a set of exercises that teach the partners to enjoy general body sensuality without intercourse. These exercises encourage a couple to enjoy body contact and sexual versatility and can help to overcome shyness which is sometimes still felt after many years of being together. Sexual intercourse is far more than a way of reproduction and includes intense emotions of attraction, love, and desire. These emotions generally begin in adolescence. When a loving bond is formed between two partners, it is important to look after that bond in any way possible. penis enlargment supplement com enhancement penis penis pump penis girth enlarement natural penis enlargment technique penis enargement herb free penis enhancement tip pennis enlargement system penis enlarement pills review penile enlargement surgery cost

From the first days of puberty and showers after physical education, boys begin to worry about the size of their penis. Older boys that are pretty endowed make fun of the younger boys that are just beginning to develop into manhood. However, this behavior does not necessarily stop when the boy is older. They still stand around and discuss aspects of their sexual life. Girls and boys, as teenagers, differ in the things they talk about when it comes to sex, men they look at the size of their penis and how far they got with their girlfriends. Girls look more to the romantic side, how cool, and good-looking their boyfriend is. Here come even more problems. To be cool the guy he must be confident, if his penis is small, he has been teased because of it and he feels inferior he will not have the confidence he needs to attract girls. This mental state travels with the boy into manhood. As a man, he still may feel inferior because of his small penis, or what he believes is a small penis because of past experiences. Women can tell a successful man with one glance, not because of his car, his clothing, or his looks but how he actually carries himself. At this point, many men with a small penis may not be performing well in the romance department, but he may also be failing at work. Men in business can see if a man has less confidence in himself and many times, he is overlooked when it comes to promotions even when he is excellent at his job. If any of this sounds familiar to you then maybe you should consider penis enlargement methods. A larger penis will give you the confidence you need to ask out the girl in the office or the one that works in the deli. You will no longer feel inferior to other men such as your boss; because you know that, he has nothing on you. Remember, though, enlarging your penis does not mean you want to get one that is so long that it scares the ladies away you just desire one that is impressive in length say around 8 inches. Longer penises can in fact be uncomfortable to some women, especially small dainty ones. Their vaginas are not long enough for the penis to fit without hitting the uterus and this can become painful with all the thrusting. Longer penises and ones with more girth with give you confidence, more stamina for a romp in the hay, more control over your ejaculation, longer love making sessions and in many cases please your lover more. Leave depression, low self-esteem, and loneliness in the past and look toward your future with a penis enlargement method that will have you on the road of success in no time. free penile enlargement pills pennis enlargement photo vimax enlargement forum free matter penis size penis enlagement without pills free penile enlargment pills penis elargement doctor penis enhancement supplement does pnis enlargement work penile enlargement surgery cost

It is true that sex is the means of reproduction of offspring. How many times you indulged in sexual encounters in those moments of passion for this reason only. Actually, most of the time pleasure and passion drive desires for having sex. Women and men have different sets of demands associated with their bodies. Achieving orgasm is the consummation of love making. Proper erection of penis and maintenance of erection till the end of intercourse are the prime requirements to take your female counterpart on new heights of pleasure. Foreplay plays vital role in igniting fire on the bed. In men it helps in erection of penis and makes it harder and elongated. Titillation on erogenous spots helps in secretion of natural lubrication in vagina for a perfect penetration. Techniques of foreplay are different. Some rely merely on kissing some other extend it upto licking. There can be stages or moments when foreplay gives no result for a man and penis refutes to become erected or it erects for a short duration. Medical science knows this problem as erectile dysfunction or simply ED. Erectile dysfunction makes a man embarrassed on the bed. Levitra can help a man who is suffering from erectile dysfunction. Penis consists of hollow capillaries. During the moments of passion, blood fills in these capillaries; this provides erection to the penis. Many neurotic and chemical changes take place during the process. One such chemical which simulates flow of blood in capillaries of penis is GMP. The chemical which inhibits GMP is phosphodiesterase type 5. This phosphodiesterase type 5 (PDE5) is responsible for taking back penis on its original position. Levitra contains Vardenafil HCl, which blocks action of PDE5 and does not let penis fall back during intercourse. It is also helpful in starting secretion of GMP so penis comes quickly in erect position. Levitra is comparatively more effective than Viagra and Cialis. Levitra can give erection upto 4 hours longer. Firm penis is what every woman love to have. She never before would have been so much sexually satisfied. Man above the age of 18 and below 65 can use Levitra. Even those males who have high cholesterol and diabetes of type1 and type2 can take Levitra. Males below 18 and women are strictly restricted from consuming Levitra. Consuming Levitra also can have some benign and short term side effects like pain in chest, swelling of limbs, problem in taking breath, pain in penis while erection. The drug can produce some allergic reactions. It should be taken strictly on doctor’s prescription. If your wife or girlfriend is always complaining that you never get prepared for second round quickly, give her surprise with many rounds of sex full of pleasure and passion. Now, it’s her turn to say no. Levitra can realize this desire.