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Genital wart signs vary from men and women, this article will explain the different genital wart signs on each person and where they appear. Genital wart signs in men tend to be more outright and seen as their genitals are completely outside the body. Genital wart signs in women seem to be more internal and not as easily seen. On women the most notable wart signs are hard dots on the outer edges of the genitals as well as on the cervix and between the vaginal opening and the anal cavity. These are the genital warts and they may not show up for a long time after contraction occurs. Men may notice that genital warts seem to show up on the head of the penis and around the anus as well as the scrotum. They may be gray or pink in color and will grow bigger over time. Another sign that a person has contracted genital warts is burning or bleeding in the genital area, especially during sexual intercourse. Genital warts may seem to appear as small white lumps which many say resemble cauliflower and can be as small as 2 millimeters. Women who have genital wart infections will notice that the warts are small, sometimes too small to see. Genital warts appear in the cervix in some cases where they cannot be seen and there are no real symptoms or signs. Except for genital warts that are internal but may bleed during sex as the warts are being torn open. Doctor’s tests will readily tell a person if she has genital warts inside her cervix. In most cases genital warts will cause no symptoms and not be noticed unless the outbreak of genital warts is big enough that a person sees or feels them. People really have to reply on the signs that genital warts gives. These are for the most part the bumps and cauliflower shapes as well as where the warts appear and how long ago a person thinks they may have gotten them. Genital warts are considered a sexually transmitted disease because they are spread during unprotected sex and in some cases protected sex. The only dangerous thing about having genital warts and not doing anything about them is if there is no discomfort, and someone chooses not to pay attention to the bumps they can lead to some cancers. pennis enlargement surgery photo cheapest penis elargement pills enargement manhattan penis penis elargement product prosolution penile enlargment pills penis elargement surgery picture penile enlargment before and after picture truth about penis enhancement pills

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Erectile dysfunction(ED) or impotence is referred to the repeated inability of a man to get or maintain an erection of penis firm enough for sexual intercourse. Due to the sensitive nature of the condition, it is difficult to estimate how many men are affected by it. Since being a very personal problem, a person suffering from ED rarely come forward to talk it openly with health personnel, but nowadays, it is discussed openly and more men are coming forward to treat it. The most common cause of Erectile dysfunction is damage to the tissues, nerves, arteries, muscles or fibrous tissue. Conditions such as diabetes, kidney disease, chronic alcoholism, multiple sclerosis and cardiovascular disease account for around 70% of cases of ED. Erectile dysfunction is also a side effect of some common medications, including blood pressure drugs, antihistamines, antidepressants and tranquilizers. According to health experts, psychological factors such as stress, anxiety, guilt, depression, low self-esteem, and fear of sexual failure cause up to 20% of cases. Smoking has also been linked to ED. Generally, people associate Erectile dysfunction with the natural process of aging. But according to several clinical studies, men of any age can be affected by this problem at some point of their sexual life. There are various ways to treat ED. Doctors will consider psychotherapy for patients who are likely to benefit, but alternatives include drugs, vacuum devices, and surgery. Most men feel embarrassed to take medication for this ailment but with the introduction of wonder drug Sildenafil citrate or Viagra in 1990s, men have come forward to treat ED. ED is treatable at any age, and awareness of this fact has been growing. More men have been seeking help and returning to normal sexual activity because of improved, successful treatments for ED. Viagra belongs to a class of drugs called phosphodiesterase inhibitors. Taken an hour before sexual activity, these drugs work by enhancing the effects of nitric oxide, a chemical that relaxes smooth muscles in the penis during sexual stimulation and allows increased blood flow. Today, with the growing use of the Internet for commercial purposes, people can order Viagra or any other anti-ED drugs from home. Buying Viagra online can protect the privacy of a buyer. The drug can be delivered to your doorsteps confidentially. Moreover, there are number of pharmaceutical companies offering generous discounts on price for those who buy Viagra online. So, if you are among those suffering from ED and feel embarrassed to discuss it to your peers, it is better to consult a doctor and buy cheap Viagra online. penis elargement cream penis enlagement pic vimax home penis enlargement pennis enlargement pills product compare penis enlargement pill natural penis enargement manual penis enlargment exercise best penile enlargment top penis enlargement pills

If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. penis enhancement secret penis enlarement excersizes enlargement penis pills vimax natural penis enlarement pills magnarx penile enlargement exercise vimax home penis enlargement surgical penis enlagement top penis enlargement pills

In ancient China, sexual practices were investigated century after century. Finally the ancient Taoist Masters began to draw conclusions about all human actions; and their conformity to ideal (Tao - the basic, eternal principle of the universe that transcends reality and is the source of being, non-being, and change) as regards the process of creation. Perhaps inspired by Tantra sexual practice, the Taoists characterized all the sexual positions, their relative advantages and drawbacks, and also even set the tone for the whole practice of the sexual act. As a creative source, the man and his penis became a subject of study in its own right. As the woman’s vagina when penetrated by the man is often exerting uneven pressure and stimulation, the Taoists developed a trusting method to stimulate, massage, and keep the man’s penis at an excellent state of readiness. The thrusting method is called the Nine Steps. To achieve this to perfection, the man must possess some amount of patience and staying power. One complete Nine Steps is 90 thrusts, so most men need training to achieve it. However, as many as possible will help the man in his general vitality and the woman partner will be ecstatic with the sexual stimulation she will receive. The Taoists also advised internalizing the ejaculation, but that is the subject of its own article. The Nine Steps Firstly, the man must find a very comfortable sexual position to execute this. One recommended position is the Horse Position. The woman lies on her back on a raised bed or table. Her legs are opened wide with the knees are pulled to her breast. The man stands in front of her, with her genitals fully exposed and open. The man then begins the Nine Steps. 1st Step. The man inserts only the head of his penis into the woman’s vagina. He makes a very shallow stroking movement exactly nine times. On the tenth stroke, he will thrust his entire penis as deep as possible. 2nd Step. Here the same is followed, but with only eight shallow penis crown strokes, and two massive whole penis thrusts thereafter. 3rd Step. Follow as above, the man will trust seven shallow times, with three hard and massive deep thrusts. 4th Step. Continuing, six shallow thrusts followed by 4 deep and profound thrusts. 5th Step. Now there are only five shallow thrusts followed by five profound ones. 6th Step. In keeping with the above, four shallow followed by six deep thrusts. 7th Step. There are only three shallow thrusts followed by seven profound ones. 8th Step. This time, only two shallow ones followed by eight deep and profound ones. 9th Step. Here at the end of the exercise, there is only one shallow thrust followed by nine rhythmic and profound thrusts. If the man was able to withstand the Nine Steps, he should rest a short while and begin again. The woman from her part may have had several orgasm! penile enlargement surgery vimax penis enlargement without pills free penis enhancement exercise vig rx penis pill penis elargement forum free penis enlarement exercise free penis enhancement exercise plastic surgery penis enlarement top penis enlargement pills

When a mother is expecting a baby for the first time she is often worried about whether she will produce enough breast milk to feed her child. This is quite natural. However some mothers still feel that they will not produce enough for their child, despite begin told not to worry from their doctor, midwife, friends and family. Perhaps, understanding how breast milk is made will alleviate those fears. During the last few months of pregnancy an expectant mother will undoubtedly begin to notice the enlargement of her breasts. Her bra cup size will increase substantially and nearing the time of birth she may feel some discomfort brought about by this welling. It's a mother's developing placenta that stimulates the release of oestrogen and progesterone, which in turn stimulate the complex biological system that makes lactation possible. The anatomy of the female breast consists of the visible area we can see on the outside; the main point of interest being the areola (nipple), and is what your new baby will latch on to. The inside tissues of the breast contain a mixture of Alveoli, ductules and ducts. These are all contained in a protective layer of fat, which is what gives women the various shapes and sizes of breasts. By the time the baby is born, glandular tissue has replaced most of the fat cells and accounts for a mother's very large breasts. Each one may get as much as 1½ pounds heavier. Milk is produced in the alveoli: A cluster of alveoli is called a lobule; a cluster of lobules is called a lobe. Each breast contains between 15 and 20 lobes, with one milk duct for every lobe. It is quite normal for an expecting mother to begin producing milk some months before the baby is born. Immediately after the baby is born, milk production goes into full swing; again this is caused by the release of hormones following the birth of the baby. A mother can expect to be producing all the milk her baby requires with 24 to 48 hours after birth. This period is called lactogenesis. Many mothers will experience painful breasts due to engorgement, although frequent feeding of baby during the first few days usually relieves this. A mother will also notice that the constuency of her breast milk is very creamy and slightly off white. Again this is normal. The milk produced during this first few days is called colostrums (or first milk). Colostrum is a high-protein, low-fat milk that is just what baby needs during its first days. It is easily digestible and packed with anti-bodies that help ward off infection. A mother's breast milk will continue to change giving just the right nutrients her baby needs during it's first year of life. Of course, in order for your baby to enjoy this goodness he will have to learn to latch on to mother's breast. This can sometimes be difficult and will need mother's help and patience. During the first days of feeding, you may feel some contractions in your abdomen as the baby sucks. The usually mild discomfort signals the release of oxytocin, which helps shrink your uterus back to its pre-pregnancy size.